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Prof.Lavanya
Vice Principal
Nehru college of Nursing
Palakkad
 Hypertension (high BP) is a disease of
vascular regulation in which the mechanisms
that control arterial pressure within the
normal range are altered. Predominant
mechanisms of control are the central
nervous system (CNS), the renal pressor
system (renin-angiotensin-aldosterone
system), and extracellular fluid volume
 Primary or Essential Hypertension
 When the diastolic pressure is 90 mm Hg
and/or the systolic pressure is 140 mm Hg or
higher and other causes of hypertension are
absent, the condition is said to be primary
hypertension.
CLASSIFICATION SBP* (MM HG)DBP* (MM HG)
Normal < 120 < 80
Prehypertension 120-139 80-89
Stage 1 hypertension 140-159 90-99
Stage 2 hypertension >160 > 100
DBP: diastolic blood pressure; SBP: systolic blood
pressure.
Joint National Committee on Prevention, Detection, Evaluation, and
Treatment of High Blood Pressure.
Systolic (mmHg) Diastolic (mmHg)
Normal blood
pressure
Less than 120 Less than 80
Elevated Between 120 and 129 Less than 80
Stage 1 hypertension Between 130 and 139 Between 80 and 89
Stage 2 hypertension At least 140 At least 90
Hypertensive crisis Over 180 Over 120
 Cause of essential hypertension is unknown;
however, there are several areas of investigation:
◦ Hyperactivity of sympathetic vasoconstricting nerves
◦ Presence of vasoactive substance released from the arterial
endothelial cells that acts on smooth muscle, sensitizing it
to vasoconstriction
◦ Increased cardiac output, followed by arteriole constriction
◦ Excessive dietary sodium intake, sodium retention, insulin
resistance, and hyperinsulinemia play roles that are not
clear
◦ Familial (genetic) tendency
 Systolic BP elevation in the absence of elevated
diastolic BP is termed isolated systolic hypertension
and is treated in the same manner.
 Occurs in approximately 5% of patients with hypertension
secondary to other pathology.
 Renal pathology:
◦ Congenital anomalies, pyelonephritis, renal artery obstruction,
acute and chronic glomerulonephritis
◦ Reduced blood flow to kidney causes release of renin. Renin
reacts with serum protein in liver (∕2-globulin) → angiotensin I;
this plus angiotensin-converting enzyme (ACE) → angiotensin II
→ leads to increased BP.
 Coarctation of aorta (stenosis of aorta) blood flow to upper
extremities is greater than flow to lower extremities
hypertension of upper part of body.
 Endocrine disturbances:
◦ Pheochromocytoma — a tumor of the adrenal gland that causes
release of epinephrine and norepinephrine and a rise in BP
(extremely rare).
◦ Adrenal cortex tumors lead to an increase in aldosterone secretion
(hyperaldosteronism) and an elevated BP (rare).
◦
Cushing's syndrome leads to an increase in adrenocortical
steroids (causing sodium and fluid retention) and hypertension.
◦ Hyperthyroidism causes increased cardiac output.
 Medications, such as estrogens, sympathomimetics,
antidepressants, NSAIDs, steroids.
 Hypertension is one of the most prevalent chronic
diseases for which treatment is available; however,
most patients with hypertension are untreated.
 There are no symptoms; thus, it is termed the silent
killer.
 Increase in incidence is associated with the following
risk factors:
◦ Age between 30 and 70
◦ Race Black
◦ Overweight, sleep apnea
◦ Family history
◦ Smoking
◦ Sedentary lifestyle
◦ Diabetes mellitus
◦ Metabolic syndrome
 Determinants of arterial pressure:
• Cardiac output
– Stroke volume: related to myocardial contractility
 and to the size of the vascular compartment.
– Heart Rate: neuronal and hormonal control
• Peripheral resistance: functional and anatomic
 changes in small arteries and arterioles
• Renin-Angiotensin-
 Aldosterone
– Vasoconstrictor properties of angiotensin II
– Sodium-retaining properties of aldosterone
 Usually
asymptomatic
 May cause
headache,
dizziness, blurred
vision when
greatly elevated
 BP readings
Headache
Ringing or buzzing in the
ears
Fatigue
Irregular heartbeat
Confusion or dizziness
Nosebleed
Blurred vision
Difficulty breathing
Chest pain
Blood in the urine
• Urinalysis for blood, protein and glucose
• Blood urea, electrolytes and creatinine
• Blood glucose
• Serum total and HDL cholesterol
• 12-lead ECG - left ventricular hypertrophy,
coronary artery disease
• Chest X-ray: cardiomegaly, heart failure,
coarctation of the aorta
• Ambulatory BP recording: assess borderline
or white coat hypertension
• Echocardiogram: detect or quantify left
ventricular hypertrophy
• Renal ultrasound: to detect possible renal
disease
• Renal angiography: detect or confirm
presence of renal artery stenosis
• Urinary catecholamines: possible
phaeochromocytoma
• Urinary cortisol and dexamethasone
suppression test: possible Cushing͛s
syndrome
• Plasma renin activity and aldosterone:
possible primary aldosteronism
 Lifestyle Modifications
 Lose weight if body mass index is greater than or equal to 25.
 Limit alcohol ” no more than 1 oz ethanol daily for men, 0.5 oz
for women.
 Get regular aerobic exercise equivalent to 30 to 45 minutes of
brisk walking most days.
 Cut sodium intake to 2.4 g or less per day.
 Include recommended daily allowances of potassium, calcium,
and magnesium in diet. This can be accomplished through
following the DASH diet (Dietary Approaches to Stop
Hypertension) rich in fruits, vegetables, low-fat dairy products,
and fiber and low in saturated and total fat.
 Smoking cessation.
 Reduce dietary saturated fat and cholesterol.
 Consider reducing coffee intake (5 cups per
day has been shown to increase BP in
hypertensive men).
 If, despite lifestyle changes, the BP remains at
or above 140/90 mm Hg (or is not at optimal
level in the presence of other cardiovascular
risk factors) over 3 to 6 months, drug therapy
should be initiated.
 If BP extremely elevated or in presence of
cardiovascular risk factors, single drug therapy
may be given.
CLASS DRUG (TRADE NAME) USUAL DOSE RANGE
IN MG/DAY* (DAILY
FREQUENCY)
SELECTED ADVERSE
EFFECTS
Thiazide
diuretics
chlorothiazide (Diuril) 125-500(1) Decreased
potassium,
sodium,
magnesium;
increased uric
acid, calcium
chlorthalidone 12.5-25(1)
hydrochlorothiazide
(Microzide, HydroDIURIL )
12.5-25 (1)
Loop
diuretics
bumetanide (Bumex) 0.5-2 (2) Decreased
potassium; short
duration of action
furosemide (Lasix) 20-80 (2)
torsemide (Demedex ) 2.5-10 (1)
Potassium-
sparing
siuretics
amiloride (Midamor ) 5-10 (1-2) Hyperkalemia
triamterene (Dyrenium) 50-100 (1-2)
Aldosterone
receptor blockers
eplerenone (Inspra) 50-100 (1-2) Hyperkalemia,
gynecomastiaspironolactone
(Aldactone)
25-50 (1-2)
Beta-adrenergic
blockers
atenolol (Tenormin) 25-100 (1) Bronchospasm,
bradycardia, heart
failure, fatigue,
hypertriglyceride
mia; may mask
hypoglycemia
betaxolol (Kerlone) 5-20 (1)
bisoprolol (Zebeta 2.5-10 (1)
metoprolol (Lopressor ) 50-100 (1-2)
Beta-adrenergic
blockers with
intrinsic
sympathomimetic
activity
acebutolol (Sectral†) 200-800 (2) Bronchospasm,
bradycardia, heart
failure, fatigue,
hypertriglyceride
mia; may mask
hypoglycemia
penbutolol (Levatol) 10-40 (1)
pindolol†10-40 (2)
Combined alpha-
and beta-
adrenergic
blockers
carvedilol (Coreg) 12.5-50 (2) Orthostatic
hypotension,
bronchospasm
labetalol (Normodyne,
Trandate†)
200-800 (2)
Angiotensin-
converting enzyme
inhibitors
captopril (Capoten†) 25-100 (2) Cough,
hyperkalemia, rash,
angioedema
enalapril (Vasotec†) 2.5-40 (1-2)
fosinopril (Monopril) 10-40 (1)
ramipril (Altace) 2.5-20 (1)
Angiotensin II
antagonists
candesartan (Atacand) 8-32 (1) Hyperkalemia,
angioedemaeprosartan (Teveten) 400-800 (1-2)
irbesartan (Avapro) 150-300 (1)
losartan (Cozaar) 25-100 (1-2)
olmesartan (Benicar) 20-40 (1)
telmisartan (Micardis) 20-80 (1)
valsartan (Diovan) 80-320 (1)
Calcium channel blockers
Non-dihydropyridines
diltiazem extended
release (Cardizem CD,
Dilacor XR, Tiazac)
180-240 (1) Conduction defects,
worsening diastole,
dysfunction,
gingival hyperplasia
diltiazem extended
release (Cardizem LA)
120-540 (1)
verapamil immediate
release (Calan, Isoptin)
80-320 (2)
Calcium channel
blockers
dihydropyridines
amlodipine (Norvasc) 2.5-10 (1) Ankle edema,
flushing,
headache
felodipine (Plendil) 2.5-20 (1)
isradipine (Dynacirc CR) 2.5-10 (2)
nicardipine sustained release
(Cardene SR)
60-120 (2)
nifedipine long-acting (Adalat
CC, Procardia XL)
30-60 (1)
Alpha1-blockers doxazosin (Cardura) 1-16 (1) Orthostatic
hypotensionprazosin (Minipress ) 2-20 (2-3)
terazosin (Hytrin) 1-20 (1-2)
Central alpha2-
agonists and other
centrally acting
drugs
clonidine (Catapres†) 0.1-0.8 (2) Sedation, dry
mouth,
bradycardia
clonidine patch (Catapres-TTS) 0.1-0.3 (1
weekly)
guanfacine 250-1000 (2)
methyldopa (Aldomet†) 0.05^-0.25 (1)
Direct vasodilators hydralazine (Apresoline) 25-100 (2) Headache, fluid
retention,
tachycardia
minoxidil (Loniten†) 2.5-80 (1-2)
• Stroke most common complication
– Cerebral haemorrhage or infarction
• Subarachnoid haemorrhage
• Hypertensive encephalopathy – rare
conditions
– High BP
– Neurological symptoms: transient disturbances
of speech or vision, paraesthesiae,
disorientation, fits and loss of consciousness.
• Neurological deficit - usually reversible if
the hypertension is properly controlled
Complications:
Central nervous system
Complications: Retina
• Optic fundi - gradation
of changes linked to the
severity of hypertension
• Cotton wool exudates
– Associated with retinal
ischaemia or infarction
– Fade in a few weeks
• Hard exudates
– Assoicated with diabetic
retinopathy
– small, white, dense
deposits of lipid
– microaneurysms ;͚dot͛
haemorrhages)
• Grade 1
• Arteriolar thickening
• Tortuosity
• Increased reflectiveness - silver
wiring
• Grade 2
• Grade 1
• Constriction of veins at arterial
crossings - arteriovenous nipping
• Grade 3
• Grade 2
• Retinal ischaemia - flame-shaped
or blot haemorrhages and ͚cotton
wool͛ exudates
• Grade 4
• Grade 3
• papilloedema
• Coronary artery disease
– Very high incidence
– Ressure load on the heart
– may lead to left ventricular hypertrophy
– forceful apex beat and fourth heart sound
• Atrial fibrillation
– diastolic dysfunction caused by left ventricular
hypertrophy
– Or the effects of coronary artery disease.
• Left ventricular failure - severe hypertension
– Absence of coronary artery disease
– Risk factor: impaired renal function, and
therefore sodium retention
• Major risk factor for renal injury and end-stage renal
disease
• Atherosclerotic, hypertension-related vascular
Lesions
- preglomerular arterioles
– Resulting in ischemic changes in the glomeruli and
postglomerular structures
– This leads to reduced GFR and, finally, a reduction in
Na and water excretion
– activation of the renin-angiotensin system
• May cause proteinuria (>3 g/24 h) or if untreated
it may cause Progressive renal failure
 Family history of high BP
 Previous episodes of high BP
 Dietary habits and salt intake
 Target organ disease or other disease processes that
may place the patient in a high-risk group diabetes,
CAD, kidney disease
 Cigarette smoking
 Episodes of headache, weakness, muscle cramp,
tingling, palpitations, sweating, vision disturbances
 Medication that could elevate BP:
◦ Hormonal contraceptives, steroids
◦ NSAIDs
◦ Nasal decongestants, appetite suppressants, tricyclic
antidepressants
 Other disease processes, such as gout, migraines,
asthma, heart failure, and benign prostatic
hyperplasia, which may be helped or worsened by
particular hypertension drugs.
 Auscultate heart rate and palpate peripheral pulses;
determine respirations.
 If skilled in doing so, perform funduscopic
examination of the eyes for the purpose of noting
vascular changes. Look for edema, spasm, and
hemorrhage of the eye vessels. Refer to
ophthalmologist for definitive diagnosis.
 Examine the heart for a shift of the point of maximal
impulse to the left, which occurs in heart
enlargement.
 Auscultate for bruits over peripheral arteries to
determine the presence of atherosclerosis, which
may be manifested as obstructed blood flow.
 Determine mentation status by asking patient about
memory, ability to concentrate, and ability to perform
simple mathematical calculations.
 Risk for Decreased Cardiac Output R/T
Increased vascular resistance,
vasoconstriction ,Myocardial ischemia
 Ventricular hypertrophy/rigidity
Nursing Interventions Rationale
Provide calm, restful surroundings, minimize
environmental activity and noise. Limit the
number of visitors and length of stay.
Helps lessen sympathetic stimulation;
promotes relaxation.
Maintain activity restrictions (bedrest or chair
rest); schedule periods of uninterrupted rest;
assist patient with self-care activities as
needed.
Lessens physical stress and tension that
affect blood pressure and the course of
hypertension.
Provide comfort measures (back and neck
massage, elevation of head).
Decreases discomfort and may reduce
sympathetic stimulation.
Instruct in relaxation techniques, guided
imagery, distractions.
Can reduce stressful stimuli, produce
calming effect, thereby reducing BP.
Monitor response to medications to control
blood pressure.
Response to drug therapy (usually
consisting of several drugs, including
diuretics, angiotensin-converting enzyme
[ACE] inhibitors, vascular smooth muscle
relaxants, beta and calcium channel
blockers) is dependent on both the
individual as well as the synergistic effects
of the drugs.Because of side effects, drug
interactions, and patient’s motivation for
taking antihypertensive medication, it is
important to use the smallest number and
lowest dosage of medications.
 Activity Intolerance R/T Generalized
weakness ,Sedentary lifestyle,
 Imbalance between oxygen supply and
demand AEB Verbal report of fatigue or
weakness,
 Abnormal heart rate or BP response to activity
 Exertional discomfort or dyspnea
 Electrocardiogram (ECG) changes reflecting
ischemia; dysrhythmias
Nursing Interventions Rationale
Assess the patient’s response to activity,
noting pulse rate more than 20 beats per
min faster than resting rate; marked
increase in BP during and after activity
(systolic pressure increase of 40 mm Hg or
diastolic pressure increase of 20 mm Hg);
dyspnea or chest pain; excessive fatigue
and weakness; diaphoresis; dizziness or
syncope.
The stated parameters are helpful in
assessing physiological responses to the
stress of activity and, if present, are
indicators of overexertion.
Assess emotional and psychological
factors affecting the current situation.
Stress or depression may be increasing the
effects of an illness, or depression might
be the result of being forced into
inactivity.
Instruct patient in energy-conserving
techniques (using chair when showering,
sitting to brush teeth or comb hair,
carrying out activities at a slower pace).
Energy-saving techniques reduce the
energy expenditure, thereby assisting in
equalization of oxygen supply and
demand.
Encourage progressive activity and self-
care when tolerated. Provide assistance as
needed.
Gradual activity progression prevents a
sudden increase in cardiac workload.
Providing assistance only as needed
encourages independence in performing
activities.
 Acute Pain Related to Increased cerebral
 vascular pressure
 verbal reports of throbbing pain located in
suboccipital region, present on awakening and
disappearing spontaneously after being up and
about
 Reluctance to move head, rubbing head,
avoidance of bright lights and noise, wrinkled
brow, clenched fists
 Changes in appetite
 Reports of stiffness of neck, dizziness, blurred
vision, nausea, and vomiting
Nursing Interventions Rationale
Encourage and maintain bed rest
during acute phase.
Minimizes stimulation and promotes
relaxation.
Provide or recommend
nonpharmacological measures for
relief of headache such as cool cloth
to forehead; back and neck rubs;
quiet, dimly lit room; relaxation
techniques (guided imagery,
distraction); and diversional
activities.
Measures that reduce cerebral
vascular pressure and that slow or
block sympathetic response are
effective in relieving headache and
associated complications.
Eliminate or minimize
vasoconstricting activities that may
aggravate headache (straining at
stool, prolonged coughing, bending
over).
Activities that increase
vasoconstriction accentuate the
headache in the presence of
increased cerebral vascular
pressure.
Assist patient with ambulation as
needed.
Dizziness and blurred vision
frequently are associated with
vascular headache. Patient may also
experience episodes of postural
hypotension, causing weakness
when ambulating.
Provide liquids, soft foods, frequent
mouth care if nosebleeds occur or
nasal packing has been done to
stop bleeding.
Promotes general comfort. Nasal
packing may interfere with
swallowing or require mouth
breathing, leading to stagnation of
oral secretions and drying of
mucous membranes.
Analgesics; Antianxiety
agents: lorazepam (Ativan),
alprazolam (Xanax), diazepam
(Valium).
Reduce or control pain and
decrease stimulation of the
sympathetic nervous system.May
aid in the reduction of tension and
discomfort that is intensified by
stress.
 Ineffective Coping R/T Situational/maturational crisis;
multiple life changes ,Inadequate relaxation; little or no
exercise, work overload
 Inadequate support systems ,Poor nutrition,Unmet
expectations; unrealistic perceptions ,Inadequate coping
methods ,Gender differences in coping strategies
 AEB Inability to meet role expectations/basic needs or
problem-solve
 Destructive behavior toward self; overeating, lack of
appetite; excessive smoking/drinking, proneness to alcohol
abuse
 Chronic fatigue/insomnia; muscular tension; frequent
head/neck aches;
 chronic worry, irritability, anxiety, emotional tension,
depression
Nursing Interventions Rationale
Assist patient to identify specific
stressors and possible strategies for
coping with them.
Recognition of stressors is the first
step in altering one’s response to the
stressor.
Include patient in planning of care,
and encourage maximum
participation in treatment plan.
Involvement provides patient with an
ongoing sense of control, improves
coping skills, and can enhance
cooperation with therapeutic regimen.
Encourage patient to evaluate life
priorities and goals. Ask questions
such as “Is what you are doing getting
you what you want?”
Focuses patient’s attention on reality
of present situation relative to
patient’s view of what is wanted.
Strong work ethic, need for “control,”
and outward focus may have led to
lack of attention to personal needs.
Assist patient to identify and begin
planning for necessary lifestyle
changes. Assist to adjust, rather than
abandon, personal/family goals.
Necessary changes should be
realistically prioritized so patient can
avoid being overwhelmed and feeling
powerless.
Help client to substitute positive
thoughts for negative ones such as ” I
can do this; I am in charge of myself.”
To provide meeting psychological
need
 Imbalanced Nutrition: More Than Body
Requirements R/T Excessive intake in relation to
metabolic need
 Sedentary activity level
 Cultural preferences AEB
 Weight 10%–20% more than ideal for height and
frame
 Triceps skinfold more than 15 mm in men and 25
mm in women (maximum for age and sex)
 Reported or observed dysfunctional eating
patterns
Nursing Interventions Rationale
Establish a realistic weight reduction plan
with the patient such as 1 lb weight loss per
wk.
Reducing caloric intake by 500 calories daily
theoretically yields a weight loss of 1 lb per
wk. Slow reduction in weight is therefore
indicative of fat loss with muscle sparing
and generally reflects a change in eating
habits.
Encourage patient to maintain a diary of food
intake, including when and where eating
takes place and the circumstances and
feelings around which the food was eaten.
Provides a database for both the adequacy of
nutrients eaten and the emotional conditions
of eating. Helps focus attention on factors
that patient has control over or can change.
Instruct and assist in appropriate food
selections, such as a diet rich in fruits,
vegetables, and low-fat dairy foods referred
to as the DASH Dietary Approaches to Stop
Hypertension) diet and avoiding foods high in
saturated fat (butter, cheese, eggs, ice cream,
meat) and cholesterol (fatty meat, egg yolks,
whole dairy products, shrimp, organ meats).
Avoiding foods high in saturated fat and
cholesterol is important in preventing
progressing atherogenesis. Moderation and
use of low-fat products in place of total
abstinence from certain food items may
prevent sense of deprivation and enhance
cooperation with dietary regimen. The DASH
diet, in conjunction with exercise, weight
loss, and limits on salt intake, may reduce or
even eliminate the need for drug therapy.
Refer to dietitian as indicated.
Can provide additional counseling and
assistance with meeting individual dietary
needs.
 Deficient Knowledge R/T
 Lack of knowledge/recall ,Information
misinterpretation ,Cognitive limitation
 Denial of diagnosis AEB
 Verbalization of the problem,Request for
information ,Statement of misconception
Nursing Interventions Rationale
Define and state the limits of desired BP. Explain
hypertension and its effects on the heart, blood
vessels, kidneys, and brain.
Provides basis for understanding elevations of BP,
and clarifies frequently used medical terminology.
Understanding that high BP can exist without
symptoms is central to enabling patient to continue
treatment, even when feeling well.
Avoid saying “normal” BP, and use the term “well-
controlled” to describe patient’s BP within desired
limits.
Because treatment for hypertension is lifelong,
conveying the idea of “control” helps patient
understand the need for continued treatment and
medication.
Assist patient in identifying modifiable risk factors
(obesity; diet high in sodium, saturated fats, and
cholesterol; sedentary lifestyle; smoking; alcohol
intake of more than 2 oz per day on a regular
basis; stressful lifestyle).
These risk factors have been shown to contribute
to hypertension and cardiovascular and renal
disease.
Problem-solve with patient to identify ways in
which appropriate lifestyle changes can be made to
reduce modifiable risk factors.
Changing “comfortable or usual” behavior patterns
can be very difficult and stressful. Support,
guidance, and empathy can enhance patient’s
success in accomplishing these tasks.
Discuss importance of eliminating smoking, and
assist patient in formulating a plan to quit
smoking.
Nicotine increases catecholamine discharge,
resulting in increased heart rate, BP,
vasoconstriction, and myocardial workload, and
reduces tissue oxygenation.
Reinforce the importance of adhering to treatment
regimen and keeping follow-up appointments.
Lack of cooperation is a common reason for failure
of antihypertensive therapy. Therefore, ongoing
evaluation for patient cooperation is critical to
successful treatment. Compliance usually improves
when patient understands causative factors and
consequences of inadequate intervention and
health maintenance.
Instruct and demonstrate technique of BP self-
monitoring. Evaluate patient’s hearing, visual
acuity, manual dexterity, and coordination.
Monitoring BP at home is reassuring to
patient because it provides visual and
positive reinforcement for efforts in
following the medical regimen and
promotes early detection of deleterious
changes.
Help patient develop a simple, convenient
schedule for taking medications.
Individualizing medication schedule to
fit patient’s personal habits and needs
may facilitate cooperation with long-
term regimen.
Explain prescribed medications along with their
rationale, dosage, expected and adverse side
effects, and idiosyncrasies
Adequate information and
understanding that side effects (mood
changes, initial weight gain, dry mouth)
are common and often subside with
time can enhance cooperation with
treatment plan.
Diuretics: Take daily doses (or larger dose) in
the early morning;
Scheduling minimizes nighttime
urination.
Weigh self on a regular schedule and record;
Primary indicator of effectiveness of
diuretic therapy.
Avoid or limit alcohol intake;
The combined vasodilating effect of
alcohol and the volume-depleting
effect of a diuretic greatly increase the
risk of orthostatic hypotension.
Notify physician if unable to tolerate
food or fluid;
Dehydration can develop rapidly if
intake is poor and patient continues
to take a diuretic.
Antihypertensives: Take prescribed
dose on a regular schedule; avoid
skipping, altering, or making up
doses; and do not discontinue
without notifying the healthcare
provider. Review potential side
effects and/or drug interactions;
Because patients often cannot feel
the difference the medication is
making in blood pressure, it is
critical that there is understanding
about the medications’ working and
side effects. For example, abruptly
discontinuing a drug may cause
rebound hypertension leading to
severe complications, or medication
may need to be altered to reduce
adverse effects.
Rise slowly from a lying to standing
position, sitting for a few minutes
before standing. Sleep with the head
slightly elevated.
Measures reduce severity of
orthostatic hypotension associated
with the use of vasodilators and
diuretics.
Suggest frequent position changes,
leg exercises when lying down.
Decreases peripheral venous pooling
that may be potentiated by
vasodilators and prolonged
sitting/standing.
Recommend avoiding hot baths,
steam rooms, and saunas,
especially with concomitant use of
alcoholic beverages.
Prevents vasodilation with potential
for dangerous side effects of
syncope and hypotension.
Instruct patient to consult
healthcare provider before taking
other prescription or over-the-
counter (OTC) medications.
Precaution is important in preventing
potentially dangerous drug
interactions. Any drug that contains
a sympathetic nervous stimulant may
increase BP or counteract
antihypertensive effects.
Instruct patient about increasing
intake of foods/ fluids high in
potassium (oranges, bananas, figs,
dates, tomatoes, potatoes, raisins,
apricots, Gatorade, and fruit juices
and foods/ fluids high in calcium
such as low-fat milk, yogurt, or
calcium supplements, as indicated).
Diuretics can deplete potassium
levels. Dietary replacement is more
palatable than drug supplements and
may be all that is needed to correct
deficit. Some studies show that 400
mg of calcium per day can lower
systolic and diastolic BP. Correcting
mineral deficiencies can also affect
BP.
Review signs and symptoms requiring notification of
healthcare provider (headache present on awakening
that does not abate; sudden and continued increase
of BP; chest pain, shortness of breath; irregular or
increased pulse rate; significant weight gain (2 lb per
day or 5 lb per wk) or peripheral and abdominal
swelling; visual disturbances; frequent, uncontrollable
nosebleeds; depression or emotional lability; severe
dizziness or episodes of fainting; muscle weakness or
cramping; nausea/ vomiting; excessive thirst.
Early detection of developing
complications, decreased
effectiveness of drug regimen or
adverse reactions to it allows for
timely intervention.
Explain rationale for prescribed dietary regimen
(usually a diet low in sodium, saturated fat, and
cholesterol).
Excess saturated fats, cholesterol,
sodium, alcohol, and calories have
been defined as nutritional risks in
hypertension. A diet low in fat and
high in polyunsaturated fat reduces
BP, possibly through prostaglandin
balance in both normotensive and
hypertensive people.
Help patient identify sources of sodium intake (table
salt, salty snacks, processed meats and cheeses,
sauerkraut, sauces, canned soups and vegetables,
baking soda, baking powder, monosodium
glutamate). Stress the importance of reading
ingredient labels of foods and OTC drugs.
Two years on a moderate low-salt
diet may be sufficient to control mild
hypertension or reduce the amount
of medication required.
Encourage patient to establish an
individual exercise program
incorporating aerobic exercise
(walking, swimming) within patient’s
capabilities. Stress the importance of
avoiding isometric activity.
Besides helping to lower BP, aerobic
activity aids in toning the
cardiovascular system. Isometric
exercise can increase serum
catecholamine levels, further elevating
BP.
Demonstrate application of ice pack to
the back of the neck and pressure
over the distal third of nose, and
recommend that patient lean the head
forward, if nosebleed occurs.
Nasal capillaries may rupture as a
result of excessive vascular pressure.
Cold and pressure constrict capillaries
to slow or halt bleeding. Leaning
forward reduces the amount of blood
that is swallowed.
Provide information regarding
community resources, and support
patient in making lifestyle changes.
Initiate referrals as indicated.
Community resources such as the
American Heart Association, “coronary
clubs,” stop smoking clinics, alcohol
(drug) rehabilitation, weight loss
programs, stress management classes,
and counseling services may be
helpful in patient’s efforts to initiate
and maintain lifestyle changes.
Hypertension

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Hypertension

  • 2.  Hypertension (high BP) is a disease of vascular regulation in which the mechanisms that control arterial pressure within the normal range are altered. Predominant mechanisms of control are the central nervous system (CNS), the renal pressor system (renin-angiotensin-aldosterone system), and extracellular fluid volume
  • 3.
  • 4.
  • 5.
  • 6.  Primary or Essential Hypertension  When the diastolic pressure is 90 mm Hg and/or the systolic pressure is 140 mm Hg or higher and other causes of hypertension are absent, the condition is said to be primary hypertension.
  • 7. CLASSIFICATION SBP* (MM HG)DBP* (MM HG) Normal < 120 < 80 Prehypertension 120-139 80-89 Stage 1 hypertension 140-159 90-99 Stage 2 hypertension >160 > 100 DBP: diastolic blood pressure; SBP: systolic blood pressure. Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure.
  • 8.
  • 9. Systolic (mmHg) Diastolic (mmHg) Normal blood pressure Less than 120 Less than 80 Elevated Between 120 and 129 Less than 80 Stage 1 hypertension Between 130 and 139 Between 80 and 89 Stage 2 hypertension At least 140 At least 90 Hypertensive crisis Over 180 Over 120
  • 10.  Cause of essential hypertension is unknown; however, there are several areas of investigation: ◦ Hyperactivity of sympathetic vasoconstricting nerves ◦ Presence of vasoactive substance released from the arterial endothelial cells that acts on smooth muscle, sensitizing it to vasoconstriction ◦ Increased cardiac output, followed by arteriole constriction ◦ Excessive dietary sodium intake, sodium retention, insulin resistance, and hyperinsulinemia play roles that are not clear ◦ Familial (genetic) tendency  Systolic BP elevation in the absence of elevated diastolic BP is termed isolated systolic hypertension and is treated in the same manner.
  • 11.  Occurs in approximately 5% of patients with hypertension secondary to other pathology.  Renal pathology: ◦ Congenital anomalies, pyelonephritis, renal artery obstruction, acute and chronic glomerulonephritis ◦ Reduced blood flow to kidney causes release of renin. Renin reacts with serum protein in liver (∕2-globulin) → angiotensin I; this plus angiotensin-converting enzyme (ACE) → angiotensin II → leads to increased BP.  Coarctation of aorta (stenosis of aorta) blood flow to upper extremities is greater than flow to lower extremities hypertension of upper part of body.
  • 12.  Endocrine disturbances: ◦ Pheochromocytoma — a tumor of the adrenal gland that causes release of epinephrine and norepinephrine and a rise in BP (extremely rare). ◦ Adrenal cortex tumors lead to an increase in aldosterone secretion (hyperaldosteronism) and an elevated BP (rare). ◦ Cushing's syndrome leads to an increase in adrenocortical steroids (causing sodium and fluid retention) and hypertension. ◦ Hyperthyroidism causes increased cardiac output.  Medications, such as estrogens, sympathomimetics, antidepressants, NSAIDs, steroids.
  • 13.
  • 14.  Hypertension is one of the most prevalent chronic diseases for which treatment is available; however, most patients with hypertension are untreated.  There are no symptoms; thus, it is termed the silent killer.  Increase in incidence is associated with the following risk factors: ◦ Age between 30 and 70 ◦ Race Black ◦ Overweight, sleep apnea ◦ Family history ◦ Smoking ◦ Sedentary lifestyle ◦ Diabetes mellitus ◦ Metabolic syndrome
  • 15.  Determinants of arterial pressure: • Cardiac output – Stroke volume: related to myocardial contractility  and to the size of the vascular compartment. – Heart Rate: neuronal and hormonal control • Peripheral resistance: functional and anatomic  changes in small arteries and arterioles
  • 16. • Renin-Angiotensin-  Aldosterone – Vasoconstrictor properties of angiotensin II – Sodium-retaining properties of aldosterone
  • 17.
  • 18.
  • 19.  Usually asymptomatic  May cause headache, dizziness, blurred vision when greatly elevated  BP readings Headache Ringing or buzzing in the ears Fatigue Irregular heartbeat Confusion or dizziness Nosebleed Blurred vision Difficulty breathing Chest pain Blood in the urine
  • 20. • Urinalysis for blood, protein and glucose • Blood urea, electrolytes and creatinine • Blood glucose • Serum total and HDL cholesterol • 12-lead ECG - left ventricular hypertrophy, coronary artery disease
  • 21. • Chest X-ray: cardiomegaly, heart failure, coarctation of the aorta • Ambulatory BP recording: assess borderline or white coat hypertension • Echocardiogram: detect or quantify left ventricular hypertrophy • Renal ultrasound: to detect possible renal disease
  • 22. • Renal angiography: detect or confirm presence of renal artery stenosis • Urinary catecholamines: possible phaeochromocytoma • Urinary cortisol and dexamethasone suppression test: possible Cushing͛s syndrome • Plasma renin activity and aldosterone: possible primary aldosteronism
  • 23.  Lifestyle Modifications  Lose weight if body mass index is greater than or equal to 25.  Limit alcohol ” no more than 1 oz ethanol daily for men, 0.5 oz for women.  Get regular aerobic exercise equivalent to 30 to 45 minutes of brisk walking most days.  Cut sodium intake to 2.4 g or less per day.  Include recommended daily allowances of potassium, calcium, and magnesium in diet. This can be accomplished through following the DASH diet (Dietary Approaches to Stop Hypertension) rich in fruits, vegetables, low-fat dairy products, and fiber and low in saturated and total fat.
  • 24.  Smoking cessation.  Reduce dietary saturated fat and cholesterol.  Consider reducing coffee intake (5 cups per day has been shown to increase BP in hypertensive men).  If, despite lifestyle changes, the BP remains at or above 140/90 mm Hg (or is not at optimal level in the presence of other cardiovascular risk factors) over 3 to 6 months, drug therapy should be initiated.  If BP extremely elevated or in presence of cardiovascular risk factors, single drug therapy may be given.
  • 25. CLASS DRUG (TRADE NAME) USUAL DOSE RANGE IN MG/DAY* (DAILY FREQUENCY) SELECTED ADVERSE EFFECTS Thiazide diuretics chlorothiazide (Diuril) 125-500(1) Decreased potassium, sodium, magnesium; increased uric acid, calcium chlorthalidone 12.5-25(1) hydrochlorothiazide (Microzide, HydroDIURIL ) 12.5-25 (1) Loop diuretics bumetanide (Bumex) 0.5-2 (2) Decreased potassium; short duration of action furosemide (Lasix) 20-80 (2) torsemide (Demedex ) 2.5-10 (1) Potassium- sparing siuretics amiloride (Midamor ) 5-10 (1-2) Hyperkalemia triamterene (Dyrenium) 50-100 (1-2)
  • 26. Aldosterone receptor blockers eplerenone (Inspra) 50-100 (1-2) Hyperkalemia, gynecomastiaspironolactone (Aldactone) 25-50 (1-2) Beta-adrenergic blockers atenolol (Tenormin) 25-100 (1) Bronchospasm, bradycardia, heart failure, fatigue, hypertriglyceride mia; may mask hypoglycemia betaxolol (Kerlone) 5-20 (1) bisoprolol (Zebeta 2.5-10 (1) metoprolol (Lopressor ) 50-100 (1-2) Beta-adrenergic blockers with intrinsic sympathomimetic activity acebutolol (Sectral†) 200-800 (2) Bronchospasm, bradycardia, heart failure, fatigue, hypertriglyceride mia; may mask hypoglycemia penbutolol (Levatol) 10-40 (1) pindolol†10-40 (2) Combined alpha- and beta- adrenergic blockers carvedilol (Coreg) 12.5-50 (2) Orthostatic hypotension, bronchospasm labetalol (Normodyne, Trandate†) 200-800 (2)
  • 27. Angiotensin- converting enzyme inhibitors captopril (Capoten†) 25-100 (2) Cough, hyperkalemia, rash, angioedema enalapril (Vasotec†) 2.5-40 (1-2) fosinopril (Monopril) 10-40 (1) ramipril (Altace) 2.5-20 (1) Angiotensin II antagonists candesartan (Atacand) 8-32 (1) Hyperkalemia, angioedemaeprosartan (Teveten) 400-800 (1-2) irbesartan (Avapro) 150-300 (1) losartan (Cozaar) 25-100 (1-2) olmesartan (Benicar) 20-40 (1) telmisartan (Micardis) 20-80 (1) valsartan (Diovan) 80-320 (1) Calcium channel blockers Non-dihydropyridines diltiazem extended release (Cardizem CD, Dilacor XR, Tiazac) 180-240 (1) Conduction defects, worsening diastole, dysfunction, gingival hyperplasia diltiazem extended release (Cardizem LA) 120-540 (1) verapamil immediate release (Calan, Isoptin) 80-320 (2)
  • 28. Calcium channel blockers dihydropyridines amlodipine (Norvasc) 2.5-10 (1) Ankle edema, flushing, headache felodipine (Plendil) 2.5-20 (1) isradipine (Dynacirc CR) 2.5-10 (2) nicardipine sustained release (Cardene SR) 60-120 (2) nifedipine long-acting (Adalat CC, Procardia XL) 30-60 (1) Alpha1-blockers doxazosin (Cardura) 1-16 (1) Orthostatic hypotensionprazosin (Minipress ) 2-20 (2-3) terazosin (Hytrin) 1-20 (1-2) Central alpha2- agonists and other centrally acting drugs clonidine (Catapres†) 0.1-0.8 (2) Sedation, dry mouth, bradycardia clonidine patch (Catapres-TTS) 0.1-0.3 (1 weekly) guanfacine 250-1000 (2) methyldopa (Aldomet†) 0.05^-0.25 (1) Direct vasodilators hydralazine (Apresoline) 25-100 (2) Headache, fluid retention, tachycardia minoxidil (Loniten†) 2.5-80 (1-2)
  • 29.
  • 30. • Stroke most common complication – Cerebral haemorrhage or infarction • Subarachnoid haemorrhage • Hypertensive encephalopathy – rare conditions – High BP – Neurological symptoms: transient disturbances of speech or vision, paraesthesiae, disorientation, fits and loss of consciousness. • Neurological deficit - usually reversible if the hypertension is properly controlled
  • 32. Complications: Retina • Optic fundi - gradation of changes linked to the severity of hypertension • Cotton wool exudates – Associated with retinal ischaemia or infarction – Fade in a few weeks • Hard exudates – Assoicated with diabetic retinopathy – small, white, dense deposits of lipid – microaneurysms ;͚dot͛ haemorrhages)
  • 33. • Grade 1 • Arteriolar thickening • Tortuosity • Increased reflectiveness - silver wiring • Grade 2 • Grade 1 • Constriction of veins at arterial crossings - arteriovenous nipping • Grade 3 • Grade 2 • Retinal ischaemia - flame-shaped or blot haemorrhages and ͚cotton wool͛ exudates • Grade 4 • Grade 3 • papilloedema
  • 34. • Coronary artery disease – Very high incidence – Ressure load on the heart – may lead to left ventricular hypertrophy – forceful apex beat and fourth heart sound • Atrial fibrillation – diastolic dysfunction caused by left ventricular hypertrophy – Or the effects of coronary artery disease. • Left ventricular failure - severe hypertension – Absence of coronary artery disease – Risk factor: impaired renal function, and therefore sodium retention
  • 35. • Major risk factor for renal injury and end-stage renal disease • Atherosclerotic, hypertension-related vascular Lesions - preglomerular arterioles – Resulting in ischemic changes in the glomeruli and postglomerular structures – This leads to reduced GFR and, finally, a reduction in Na and water excretion – activation of the renin-angiotensin system • May cause proteinuria (>3 g/24 h) or if untreated it may cause Progressive renal failure
  • 36.  Family history of high BP  Previous episodes of high BP  Dietary habits and salt intake  Target organ disease or other disease processes that may place the patient in a high-risk group diabetes, CAD, kidney disease  Cigarette smoking  Episodes of headache, weakness, muscle cramp, tingling, palpitations, sweating, vision disturbances  Medication that could elevate BP: ◦ Hormonal contraceptives, steroids ◦ NSAIDs ◦ Nasal decongestants, appetite suppressants, tricyclic antidepressants  Other disease processes, such as gout, migraines, asthma, heart failure, and benign prostatic hyperplasia, which may be helped or worsened by particular hypertension drugs.
  • 37.  Auscultate heart rate and palpate peripheral pulses; determine respirations.  If skilled in doing so, perform funduscopic examination of the eyes for the purpose of noting vascular changes. Look for edema, spasm, and hemorrhage of the eye vessels. Refer to ophthalmologist for definitive diagnosis.  Examine the heart for a shift of the point of maximal impulse to the left, which occurs in heart enlargement.  Auscultate for bruits over peripheral arteries to determine the presence of atherosclerosis, which may be manifested as obstructed blood flow.  Determine mentation status by asking patient about memory, ability to concentrate, and ability to perform simple mathematical calculations.
  • 38.  Risk for Decreased Cardiac Output R/T Increased vascular resistance, vasoconstriction ,Myocardial ischemia  Ventricular hypertrophy/rigidity
  • 39. Nursing Interventions Rationale Provide calm, restful surroundings, minimize environmental activity and noise. Limit the number of visitors and length of stay. Helps lessen sympathetic stimulation; promotes relaxation. Maintain activity restrictions (bedrest or chair rest); schedule periods of uninterrupted rest; assist patient with self-care activities as needed. Lessens physical stress and tension that affect blood pressure and the course of hypertension. Provide comfort measures (back and neck massage, elevation of head). Decreases discomfort and may reduce sympathetic stimulation. Instruct in relaxation techniques, guided imagery, distractions. Can reduce stressful stimuli, produce calming effect, thereby reducing BP. Monitor response to medications to control blood pressure. Response to drug therapy (usually consisting of several drugs, including diuretics, angiotensin-converting enzyme [ACE] inhibitors, vascular smooth muscle relaxants, beta and calcium channel blockers) is dependent on both the individual as well as the synergistic effects of the drugs.Because of side effects, drug interactions, and patient’s motivation for taking antihypertensive medication, it is important to use the smallest number and lowest dosage of medications.
  • 40.  Activity Intolerance R/T Generalized weakness ,Sedentary lifestyle,  Imbalance between oxygen supply and demand AEB Verbal report of fatigue or weakness,  Abnormal heart rate or BP response to activity  Exertional discomfort or dyspnea  Electrocardiogram (ECG) changes reflecting ischemia; dysrhythmias
  • 41. Nursing Interventions Rationale Assess the patient’s response to activity, noting pulse rate more than 20 beats per min faster than resting rate; marked increase in BP during and after activity (systolic pressure increase of 40 mm Hg or diastolic pressure increase of 20 mm Hg); dyspnea or chest pain; excessive fatigue and weakness; diaphoresis; dizziness or syncope. The stated parameters are helpful in assessing physiological responses to the stress of activity and, if present, are indicators of overexertion. Assess emotional and psychological factors affecting the current situation. Stress or depression may be increasing the effects of an illness, or depression might be the result of being forced into inactivity. Instruct patient in energy-conserving techniques (using chair when showering, sitting to brush teeth or comb hair, carrying out activities at a slower pace). Energy-saving techniques reduce the energy expenditure, thereby assisting in equalization of oxygen supply and demand. Encourage progressive activity and self- care when tolerated. Provide assistance as needed. Gradual activity progression prevents a sudden increase in cardiac workload. Providing assistance only as needed encourages independence in performing activities.
  • 42.  Acute Pain Related to Increased cerebral  vascular pressure  verbal reports of throbbing pain located in suboccipital region, present on awakening and disappearing spontaneously after being up and about  Reluctance to move head, rubbing head, avoidance of bright lights and noise, wrinkled brow, clenched fists  Changes in appetite  Reports of stiffness of neck, dizziness, blurred vision, nausea, and vomiting
  • 43. Nursing Interventions Rationale Encourage and maintain bed rest during acute phase. Minimizes stimulation and promotes relaxation. Provide or recommend nonpharmacological measures for relief of headache such as cool cloth to forehead; back and neck rubs; quiet, dimly lit room; relaxation techniques (guided imagery, distraction); and diversional activities. Measures that reduce cerebral vascular pressure and that slow or block sympathetic response are effective in relieving headache and associated complications. Eliminate or minimize vasoconstricting activities that may aggravate headache (straining at stool, prolonged coughing, bending over). Activities that increase vasoconstriction accentuate the headache in the presence of increased cerebral vascular pressure.
  • 44. Assist patient with ambulation as needed. Dizziness and blurred vision frequently are associated with vascular headache. Patient may also experience episodes of postural hypotension, causing weakness when ambulating. Provide liquids, soft foods, frequent mouth care if nosebleeds occur or nasal packing has been done to stop bleeding. Promotes general comfort. Nasal packing may interfere with swallowing or require mouth breathing, leading to stagnation of oral secretions and drying of mucous membranes. Analgesics; Antianxiety agents: lorazepam (Ativan), alprazolam (Xanax), diazepam (Valium). Reduce or control pain and decrease stimulation of the sympathetic nervous system.May aid in the reduction of tension and discomfort that is intensified by stress.
  • 45.  Ineffective Coping R/T Situational/maturational crisis; multiple life changes ,Inadequate relaxation; little or no exercise, work overload  Inadequate support systems ,Poor nutrition,Unmet expectations; unrealistic perceptions ,Inadequate coping methods ,Gender differences in coping strategies  AEB Inability to meet role expectations/basic needs or problem-solve  Destructive behavior toward self; overeating, lack of appetite; excessive smoking/drinking, proneness to alcohol abuse  Chronic fatigue/insomnia; muscular tension; frequent head/neck aches;  chronic worry, irritability, anxiety, emotional tension, depression
  • 46. Nursing Interventions Rationale Assist patient to identify specific stressors and possible strategies for coping with them. Recognition of stressors is the first step in altering one’s response to the stressor. Include patient in planning of care, and encourage maximum participation in treatment plan. Involvement provides patient with an ongoing sense of control, improves coping skills, and can enhance cooperation with therapeutic regimen. Encourage patient to evaluate life priorities and goals. Ask questions such as “Is what you are doing getting you what you want?” Focuses patient’s attention on reality of present situation relative to patient’s view of what is wanted. Strong work ethic, need for “control,” and outward focus may have led to lack of attention to personal needs. Assist patient to identify and begin planning for necessary lifestyle changes. Assist to adjust, rather than abandon, personal/family goals. Necessary changes should be realistically prioritized so patient can avoid being overwhelmed and feeling powerless. Help client to substitute positive thoughts for negative ones such as ” I can do this; I am in charge of myself.” To provide meeting psychological need
  • 47.  Imbalanced Nutrition: More Than Body Requirements R/T Excessive intake in relation to metabolic need  Sedentary activity level  Cultural preferences AEB  Weight 10%–20% more than ideal for height and frame  Triceps skinfold more than 15 mm in men and 25 mm in women (maximum for age and sex)  Reported or observed dysfunctional eating patterns
  • 48. Nursing Interventions Rationale Establish a realistic weight reduction plan with the patient such as 1 lb weight loss per wk. Reducing caloric intake by 500 calories daily theoretically yields a weight loss of 1 lb per wk. Slow reduction in weight is therefore indicative of fat loss with muscle sparing and generally reflects a change in eating habits. Encourage patient to maintain a diary of food intake, including when and where eating takes place and the circumstances and feelings around which the food was eaten. Provides a database for both the adequacy of nutrients eaten and the emotional conditions of eating. Helps focus attention on factors that patient has control over or can change. Instruct and assist in appropriate food selections, such as a diet rich in fruits, vegetables, and low-fat dairy foods referred to as the DASH Dietary Approaches to Stop Hypertension) diet and avoiding foods high in saturated fat (butter, cheese, eggs, ice cream, meat) and cholesterol (fatty meat, egg yolks, whole dairy products, shrimp, organ meats). Avoiding foods high in saturated fat and cholesterol is important in preventing progressing atherogenesis. Moderation and use of low-fat products in place of total abstinence from certain food items may prevent sense of deprivation and enhance cooperation with dietary regimen. The DASH diet, in conjunction with exercise, weight loss, and limits on salt intake, may reduce or even eliminate the need for drug therapy. Refer to dietitian as indicated. Can provide additional counseling and assistance with meeting individual dietary needs.
  • 49.  Deficient Knowledge R/T  Lack of knowledge/recall ,Information misinterpretation ,Cognitive limitation  Denial of diagnosis AEB  Verbalization of the problem,Request for information ,Statement of misconception
  • 50. Nursing Interventions Rationale Define and state the limits of desired BP. Explain hypertension and its effects on the heart, blood vessels, kidneys, and brain. Provides basis for understanding elevations of BP, and clarifies frequently used medical terminology. Understanding that high BP can exist without symptoms is central to enabling patient to continue treatment, even when feeling well. Avoid saying “normal” BP, and use the term “well- controlled” to describe patient’s BP within desired limits. Because treatment for hypertension is lifelong, conveying the idea of “control” helps patient understand the need for continued treatment and medication. Assist patient in identifying modifiable risk factors (obesity; diet high in sodium, saturated fats, and cholesterol; sedentary lifestyle; smoking; alcohol intake of more than 2 oz per day on a regular basis; stressful lifestyle). These risk factors have been shown to contribute to hypertension and cardiovascular and renal disease. Problem-solve with patient to identify ways in which appropriate lifestyle changes can be made to reduce modifiable risk factors. Changing “comfortable or usual” behavior patterns can be very difficult and stressful. Support, guidance, and empathy can enhance patient’s success in accomplishing these tasks. Discuss importance of eliminating smoking, and assist patient in formulating a plan to quit smoking. Nicotine increases catecholamine discharge, resulting in increased heart rate, BP, vasoconstriction, and myocardial workload, and reduces tissue oxygenation. Reinforce the importance of adhering to treatment regimen and keeping follow-up appointments. Lack of cooperation is a common reason for failure of antihypertensive therapy. Therefore, ongoing evaluation for patient cooperation is critical to successful treatment. Compliance usually improves when patient understands causative factors and consequences of inadequate intervention and health maintenance.
  • 51. Instruct and demonstrate technique of BP self- monitoring. Evaluate patient’s hearing, visual acuity, manual dexterity, and coordination. Monitoring BP at home is reassuring to patient because it provides visual and positive reinforcement for efforts in following the medical regimen and promotes early detection of deleterious changes. Help patient develop a simple, convenient schedule for taking medications. Individualizing medication schedule to fit patient’s personal habits and needs may facilitate cooperation with long- term regimen. Explain prescribed medications along with their rationale, dosage, expected and adverse side effects, and idiosyncrasies Adequate information and understanding that side effects (mood changes, initial weight gain, dry mouth) are common and often subside with time can enhance cooperation with treatment plan. Diuretics: Take daily doses (or larger dose) in the early morning; Scheduling minimizes nighttime urination. Weigh self on a regular schedule and record; Primary indicator of effectiveness of diuretic therapy. Avoid or limit alcohol intake; The combined vasodilating effect of alcohol and the volume-depleting effect of a diuretic greatly increase the risk of orthostatic hypotension.
  • 52. Notify physician if unable to tolerate food or fluid; Dehydration can develop rapidly if intake is poor and patient continues to take a diuretic. Antihypertensives: Take prescribed dose on a regular schedule; avoid skipping, altering, or making up doses; and do not discontinue without notifying the healthcare provider. Review potential side effects and/or drug interactions; Because patients often cannot feel the difference the medication is making in blood pressure, it is critical that there is understanding about the medications’ working and side effects. For example, abruptly discontinuing a drug may cause rebound hypertension leading to severe complications, or medication may need to be altered to reduce adverse effects. Rise slowly from a lying to standing position, sitting for a few minutes before standing. Sleep with the head slightly elevated. Measures reduce severity of orthostatic hypotension associated with the use of vasodilators and diuretics. Suggest frequent position changes, leg exercises when lying down. Decreases peripheral venous pooling that may be potentiated by vasodilators and prolonged sitting/standing.
  • 53. Recommend avoiding hot baths, steam rooms, and saunas, especially with concomitant use of alcoholic beverages. Prevents vasodilation with potential for dangerous side effects of syncope and hypotension. Instruct patient to consult healthcare provider before taking other prescription or over-the- counter (OTC) medications. Precaution is important in preventing potentially dangerous drug interactions. Any drug that contains a sympathetic nervous stimulant may increase BP or counteract antihypertensive effects. Instruct patient about increasing intake of foods/ fluids high in potassium (oranges, bananas, figs, dates, tomatoes, potatoes, raisins, apricots, Gatorade, and fruit juices and foods/ fluids high in calcium such as low-fat milk, yogurt, or calcium supplements, as indicated). Diuretics can deplete potassium levels. Dietary replacement is more palatable than drug supplements and may be all that is needed to correct deficit. Some studies show that 400 mg of calcium per day can lower systolic and diastolic BP. Correcting mineral deficiencies can also affect BP.
  • 54. Review signs and symptoms requiring notification of healthcare provider (headache present on awakening that does not abate; sudden and continued increase of BP; chest pain, shortness of breath; irregular or increased pulse rate; significant weight gain (2 lb per day or 5 lb per wk) or peripheral and abdominal swelling; visual disturbances; frequent, uncontrollable nosebleeds; depression or emotional lability; severe dizziness or episodes of fainting; muscle weakness or cramping; nausea/ vomiting; excessive thirst. Early detection of developing complications, decreased effectiveness of drug regimen or adverse reactions to it allows for timely intervention. Explain rationale for prescribed dietary regimen (usually a diet low in sodium, saturated fat, and cholesterol). Excess saturated fats, cholesterol, sodium, alcohol, and calories have been defined as nutritional risks in hypertension. A diet low in fat and high in polyunsaturated fat reduces BP, possibly through prostaglandin balance in both normotensive and hypertensive people. Help patient identify sources of sodium intake (table salt, salty snacks, processed meats and cheeses, sauerkraut, sauces, canned soups and vegetables, baking soda, baking powder, monosodium glutamate). Stress the importance of reading ingredient labels of foods and OTC drugs. Two years on a moderate low-salt diet may be sufficient to control mild hypertension or reduce the amount of medication required.
  • 55. Encourage patient to establish an individual exercise program incorporating aerobic exercise (walking, swimming) within patient’s capabilities. Stress the importance of avoiding isometric activity. Besides helping to lower BP, aerobic activity aids in toning the cardiovascular system. Isometric exercise can increase serum catecholamine levels, further elevating BP. Demonstrate application of ice pack to the back of the neck and pressure over the distal third of nose, and recommend that patient lean the head forward, if nosebleed occurs. Nasal capillaries may rupture as a result of excessive vascular pressure. Cold and pressure constrict capillaries to slow or halt bleeding. Leaning forward reduces the amount of blood that is swallowed. Provide information regarding community resources, and support patient in making lifestyle changes. Initiate referrals as indicated. Community resources such as the American Heart Association, “coronary clubs,” stop smoking clinics, alcohol (drug) rehabilitation, weight loss programs, stress management classes, and counseling services may be helpful in patient’s efforts to initiate and maintain lifestyle changes.
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